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Immune Competence and Blood Lipids

 

This eleven minute documentary explains why over 90% of people who take food based immune modulators normalize cholesterol and triglycerides 30 days.

The observation that immune competence strongly influences blood lipids is very important for millions of Americans with elevated heart disease risk and health care providers.

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See clinical trial results here. In summary:

  • 30 of 31 cases of elevated triglycerides normalized in 30 days;
  • 26 of 28 cases of elevated cholesterol normalized in 30 days.

Clinical studies show surprising clinical efficacy for treatment of high cholesterol with mushroom based heteropolysaccharides.

Clinical data showing food based  heteropolysaccharides are more effective than statin drugs for cholesterol management is huge news for millions of Americans who prefer to manage vascular and heart disease risk without drugs, side effects or expensive life-long prescriptions.

From a clinical viewpoint, doctors don't even dream of thinking about about the immune system when they treat heart disease. It's like relating clogged pipes with the weather. The two obviously have nothing to do with each other, or do they?

Why would immune competence change high cholesterol? This relationship suggests that there is an intimate relationship between immune competence and heart disease, the second leading cause of death in North America. It is fascinating that over 90% of this study's participants normalized both cholesterol and triglycerides within 30 days using only immune system modulators.

This is a huge surprise, just like when we found out that peptic ulcers were an infection. Barry Marshall and Robin Warren got a Nobel Prize in 2005 for discovery that Peptic ulcers are caused when helicobacter pylori bacteria. The bacteria in the stomach lining caused stomach lesions. From a humanitarian view, nearly everybody who had suffered with ulcers, sometimes for decades, could be cured for $10 worth of antibiotics. That's a big deal with a huge humanitarian impact.

This data correlates immune competence with vascular disease. The data indicates that enhanced immune competence quickly reduces the risk markers for cardiovascular disease.

This is huge because it affects nearly everyone. Vascular, and heart disease have a 30 year running history killing more Americans than any other disease. Any study that tells us why so many are sick, and suggests that it's may be as simple as fortifying the body against infectious disease is a huge contribution.

This data suggests that doctors should evaluate immune competence for anyone who has high cholesterol and provide immune modulators for patients with any evidence of systemic inflammation, elevated C reactive protein, or any other inflammatory marker. Inflammatory markers nearly always accompany elevated cholesterol.

Wouldn't it be fascinating if hypercholesterolemia, high cholesterol, high triglycerides, were all symptoms of a systemic infection? Wouldn't it be amazing if vascular disease was accumulated result of a chronic infection?

Well. That's what the data says.

But Why?

Conventional medicine associates hyperlipidemia with fat consumption and lack of exercise. This causal model mismatches both observed and and research data, most notably in the failure of cholesterol drugs to decrease the observation of adverse cardiovascular events in patients.

While it is true that Individuals with low cholesterol have lower risk of cardiovascular disease. Drugs that suppress cholesterol production do not appear to inhibit the progression of vascular disease. Conversely, lifestyle shifts that reduce cholesterol inhibit or halt vascular disease progression. So why the difference? Why doesn't artificial cholesterol suppression prevent future cardiovascular disease?

A simple explanation. Elevated blood lipids don't cause cardiovascular disease. They're are a symptom. Suppressing blood lipids, manipulates the symptom without addressing the cause, and creates canceling effects:

  • a) it reduces plaque accumulation by reducing availability of LDL cholesterol;
  • b) it inhibits beneficial cell reconstruction by reducing availability of cellular building materials;
  • c) the two factors cancel each other out, leaving the body in prolonged disrepair;
  • d) with cause unresolved.

Conversely, lifestyle and diet changes, which result in lowered cholesterol overcome the cause of the elevated lipids and eliminate the ongoing need to produce extra lipids for healing. Resulting in lower cholesterol.

The body's inability to overcome a systemic infection is the misidentified cause of elevated blood lipids, and the dominant risk factor in cardiovascular disease. This explains contrasting positive benefits of lifestyle modification versus to neutral and negative results from artificial lipid modulation.

Strong documentation relates pathogenic agents, and cardiovascular disease, diabetes, cancer. The pathogenic connection provides a plausible explanation why nonspecific immunotherapy is strikingly effective in reducing key risk markers for cardiovascular disease:

Cardiovascular disease is caused by disease agents kept in check by a healthy immune system.

These books describe pathogenic-disease models for diabetes, heart disease, and cancer:

The Connection Chain

  • Lipids and cholesterol are critical building blocks for cells. Each cell membrane is comprised of proteins, glycolipids and phospholipids with a majority of structure provided by phospholipids and cholesterol;
  • The blood is a carrying system. In hyperlipidemia and high cholesterol , the blood carries elevated amounts of fat-based or lipid building materials throughout the body to repair cell membranes damaged by pathogenic attack;
  • Pathogens damage the cell membranes, through a variety of means, creating ongoing elevated demand for membrane building materials and elevating blood lipids;
  • The liver creates lipids and puts them into the blood to repair ongoing damage.

First Hypothesis:

  • Hyperlipidemia is a symptom of systemic infection;
  • Which damages cell membranes;
  • Causing the liver to provide an elevated supply of cell membrane lipid materials;
  • Which are detected in the blood as hyperlipidemia.

Second Hypothesis:

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Keywords: hyperlipidemia, hypercholesterolemia, immune competence, high cholesterol, high triglycerides, immune competence

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