Background
Systemic alkalosis reflects a systemic acid deficiency caused by pathgogenic, toxins, or traumatic stress which drives elevated cellular use of chlorine, and eventually depletion.
This protocol is based on Revici model of the toxin/chlorine metabolism response process:
- The body produces specialized fatty acids as an initial response to stress or shock;
- These agents collect and persist in cells and assert persistent resistive influence to the stress influence;
- The body breaks down these agents over time. Breakdown utilizes a spectrum of elements including chlorine, bivalent negative sulfur, and selenium.
- Breadown often depletes thes metabolic agents.
- Chronic depletion of these agents often further contributes to the disease process.
Many conditions, including Viral infection, create systemic alkalosis. Alkalosis is indicated when the weighted average of saliva and urine pH exceed 6.4. To calculate the weighted average multiply saliva pH by 2 and add Urine pH and divide by 3. This is the weighted average.
Alkalosis contributes a metabolic challenge because it is a frequent cofactor in toxin accumulation, digestive dysfunction and tissue syptomes. I starts out as compensation indicator and when chronically unaddressed, becomes a dominant, and eventually dangerous, factor in persistent health issues.
This imbalance indicates an absence of stomach acid which harmfully limits both digestive nutrient absorption and limits the livers abiliy to release bile, and hence detoxify the body.
This imbalance is frequently observed in a variety of conditions:
- Colds and Flu
- Cancer
- Spectral Pathogen Infections (Lyme Disease)
|